ACUTE GLOMERULONEPHRITIS (GN)
Historically called the “nephritic syndrome.” Characterized by development, over days, of azotemia, hypertension, edema, hematuria, proteinuria, and sometimes oliguria. Salt and water retention are due to reduced glomerular filtration rate (GFR) and may result in circulatory congestion. Red blood cell (RBC) casts on urinalysis confirm diagnosis. Proteinuria is usually <3 g/d. Most forms of acute GN are mediated by humoral immune mechanisms. Clinical course depends on underlying lesion (Table 145-1).
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aMost common cause.
Source: Glassock RJ, Brenner BM: Harrison’s Principles of Internal Medicine, 13th ed, 1995.
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Citation
Kasper, Dennis L., et al., editors. "ACUTE GLOMERULONEPHRITIS (GN)." Harrison's Manual of Medicine, 20th ed., McGraw Hill Inc., 2020. harrisons.unboundmedicine.com/harrisons/view/Harrisons-Manual-of-Medicine/623344/all/ACUTE_GLOMERULONEPHRITIS__GN_.
ACUTE GLOMERULONEPHRITIS (GN). In: Kasper DLD, Fauci ASA, Hauser SLS, et al, eds. Harrison's Manual of Medicine. McGraw Hill Inc.; 2020. https://harrisons.unboundmedicine.com/harrisons/view/Harrisons-Manual-of-Medicine/623344/all/ACUTE_GLOMERULONEPHRITIS__GN_. Accessed November 10, 2024.
ACUTE GLOMERULONEPHRITIS (GN). (2020). In Kasper, D. L., Fauci, A. S., Hauser, S. L., Longo, D. L., Jameson, J. L., & Loscalzo, J. (Eds.), Harrison's Manual of Medicine (20th ed.). McGraw Hill Inc.. https://harrisons.unboundmedicine.com/harrisons/view/Harrisons-Manual-of-Medicine/623344/all/ACUTE_GLOMERULONEPHRITIS__GN_
ACUTE GLOMERULONEPHRITIS (GN) [Internet]. In: Kasper DLD, Fauci ASA, Hauser SLS, Longo DLD, Jameson JLJ, Loscalzo JJ, editors. Harrison's Manual of Medicine. McGraw Hill Inc.; 2020. [cited 2024 November 10]. Available from: https://harrisons.unboundmedicine.com/harrisons/view/Harrisons-Manual-of-Medicine/623344/all/ACUTE_GLOMERULONEPHRITIS__GN_.
* Article titles in AMA citation format should be in sentence-case
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